Host-Directed Antiviral Research Center, College of Veterinary Medicine, Chonnam National University

- 2010. 3. – 2013. 2.: Ph.D. in Veterinary Pharmacology and Toxicology, College of Veterinary Medicine, Chonnam National University, South Korea.

- 2008. 3. – 2010. 2.: M.S. in Veterinary Pharmacology and Toxicology, College of Veterinary Medicine, Chonnam National University, South Korea.

- 1998. 3. – 2004. 2.: Bachelor in Veterinary Medicine, College of Veterinary Medicine, Chonnam National University, South Korea.

- 2010-2012: Researcher, Korea Institute of Oriental Medicine

- 2013-2014: Post-doctor, Korea Research Institute of Bioscience and Biotechnology

- 2015-present: Associated professor, College of Veterinary Medicine, Chonnam National University

1. Kim YH, et al. 2023. Protease allergen-induced HMGB1 contributes to NLRC4 inflammasome mediated inflammation in experimental asthma. Allergy 78: 1387-1392. (Corresponding author, IF=12.4)

2. Park SW, et al. 2022. Loranthus tanakae Franch. & Sav. suppresses inflammatory response in cigarette smoke condensate exposed bronchial epithelial cells and mice. Antioxidants 11: 1885. (Corresponding author, IF=7.0)

3. Pak SW, et al. 2022. Anti-asthmatic effects of Phlomis umbrosa Turczaninow using ovalbumin induced asthma murine model and network pharmacology analysis. Biomed. Pharmacother. 145: 112410. (Corresponding author, IF=7.5) 

4. Shin NR, et al. 2020. Scrophularia buergeriana attenuates allergic inflammation by reducing NF-kB activation. Phytomedicine 67: 153159. (Corresponding author, IF=7.9)

5. Ko JW, et al. 2020. 4-hydroxycinnamic acid suppresses airway inflammation and mucus hypersecretion in allergic asthma induced by ovalbumin challenge. Phytother. Res. 34: 624-633. (Corresponding author, IF=7.2)

6. Lim JO, et al. 2019. Cisplatin-induced ototoxicity involves interaction of PRMT3 and cannabinoid system. Arch. Toxicol. 93: 2335-2346. (Corresponding author, IF=6.1)

Respiratory Pharmacology: My major interest is to study the molecular pathogenesis of respiratory disease including allergic asthma, chronic obstructive pulmonary disease and pulmonary fibrosis. The current emphasis is on identifying the mechanism of action of respiratory damage induced by fine dust, and additional research is being conducted on the correlation between fine dust exposure and the occurrence of underlying respiratory diseases.